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If I were to take a single faecal sample from each of the same individuals and performed massive, parallel, high throughput, 454-sequencing to generate sequence lists, relative abundances of different phylogenetic groups and pie-charts, and used these data to postulate continent associated differences in intestinal microbial communities, it is likely that the paper would be published in one of the highest profile, general science journals. Statistical analysis and modelling of gene count data in metagenomics.

There are already several precedents for this and, as a community, we should be embarrassed.' Seven years on from the above comment about this predicament, little has changed regarding this status quo even as microbiome research has vastly expanded in scope, entering any and all areas of research on human physiology and disease.

But in certain types of cancer this enzyme actually enhances tumor survival and undermines chemotherapies designed to damage DNA in cancer cells.

A recent human clinical trial found that drugs that inhibited PARP-1 reduced tumor growth in breast-cancer patients with mutations in certain DNA-repair (BRCA-1 and BRCA-2) genes.

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So we could be looking at an isolated haplogroup below DF13 that existed for ~500-1,000 years but was reduced to a very low then had rapid expansion with a lot of sons.

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is a well-documented risk factor for several forms of gastric cancer, but researchers have not yet determined the mechanisms by which specific bacterial factors contribute to cancer development.

Nearly one-half of the world's population is infected with , is the first to show that a factor produced by the bacterium directly activates poly(ADP-ribose) polymerase-1 (PARP-1), an enzyme found primarily within the nucleus of animal cells.

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Testing is under way to confirm it but it appears that the Big Y testing misses 2 of the 5 main Patriarch SNP's found under DF13.